Eye Movement Desensitization Reprocessing, or EMDR, was first discovered in 1987 by Psychologist Francis Shapiro.  One day, she felt anxious by negative thoughts, so went for a walk in the woods. Sure, you might say, sounds like a great way to shed some stress – walk it off. But as she moved her eyes from right to left, taking in nature’s greens, yellows, and browns, she noticed that her level of anxiety decreased. She tried a similar eye movement process with her clients, and it worked. They, too, experienced a reduction in anxiety. That’s how EMDR was born. Initially used to treat individuals with PTSD and other anxiety disorders, it is now used to treat other conditions like depression, schizophrenia and eating disorders.

When you cut your finger, cells race to the site and clump together to stop the bleeding. Your body’s nutrients then heal the wound. If it continues to get irritated, it will become inflamed and may open up again. Once the source of irritation is removed, the wound can finally heal. PTSD symptoms are like festering wounds; the goal of EMDR is to remove what is blocking those wounds from healing.

Here’s how EMDR works: The client recalls a vivid image from the trauma experienced, such as a lying on the pavement after being hit by a car while thinking about a negative belief about the self, such as “I’m going to die.” The client notes the visceral sensations and emotions she is experiencing like a tight chest, shortness of breath and increased fear. She then thinks of a positive image like taking a warm bath, then a thought: “I’m safe.” Following this, the client conjures the negative thoughts and images while undergoing 15-20 seconds of bilateral stimulation: lateral eye movements, where the client follows the back and forth movement of the therapist’s fingers. Even though lateral eye movements are the most common form of stimulation, other stimuli may be just as effective, such as tapping or tones. The therapist who treated me used walkie-talkie like devices that vibrated beneath the backs of my thighs.

The same bilateral stimulation is then used while the client focuses on the positive images and thoughts. Over time, these will become embedded in the client’s memory, blocking the negative images and thoughts – the wounds. Eventually, PTSD symptoms will lessen when exposed to sounds, smells, or sights reminiscent of a trauma or threat.

EMDR is different from exposure therapy, which involves prolonged exposure to a stimulus that triggers thoughts, emotions, and sensations about the traumatic event. Some therapists believe that prolonged exposure is necessary to produce effective treatment outcomes. But others believe that this type of therapy will cause fearful memories to outweigh the joyful ones in people who have experienced extreme trauma, then encounter a particularly stressful situation in the future.

http://www.emdr.com

How do you predict who will suffer from PTSD after a traumatic event and who will not? Most people who experience a traumatic event actually do not suffer from PTSD: about 56% of people will experience a traumatic event in their lifetime, but only 8% will develop PTSD.

Research studies show that individuals with a variant of two genes – TPH1 and TPH2 – are more likely to develop symptoms. These genes, which control levels of serotonin – a chemical in the nervous system that regulates mood, sleep, and alertness – are altered in PTSD sufferers.

Genetics aside, other factors increase the risk for PTSD:

Having experienced other trauma earlier in life, including childhood abuse or neglect.

Having other mental health problems, such as anxiety or depression Lacking a good support system of family and friends Having biological (blood) relatives with mental health problems, including PTSD or depression.

Gender: Because there is more societal pressure on females to take care of others, we are twice as likely than men to suffer from anxiety disorders, such as PTSD.

Personality: People who are worriers, and cannot tolerate unpredictability. These traits may have a biological basis. It’s possible that the amygdala, the part of the brain that controls emotion, is oversensitive in worriers.

Have you been diagnosed with PTSD? If so, it may come as a relief to know that you can blame your symptoms on factors beyond your control.

http://www.ncbi.nlm.nih.gov/books/NBK49142

http://www.mayoclinic.org/diseases-conditions/post-traumatic-stress-disorder/basics/risk-factors/con-20022540

http://www.nimh.nih.gov/statistics/1AD_PTSD_ADULT.shtml

http://newsroom.ucla.edu/releases/ucla-study-identifies-first-genes-231248

http://psychnews.psychiatryonline.org/newsArticle.aspx?articleid=1130400

When exposed to danger, it’s natural to be afraid. Our bodies are triggered to make a split-second decision to either face the danger, or run from it: the “flight-or-fight” response. This is a healthy reaction. But in those with PTSD, they continue to be afraid and feel stressed long after the danger has passed – in my case, the speeding car at the Santa Monica Farmers’ Market. Symptoms, like avoiding places that trigger memories of the event, nightmares, depression, and hyper-vigilance – heightened awareness of your surroundings – may interfere with day-to-day-life. In hyper-vigilance, there is a perpetual scanning of the environment for sights, sounds, smells, or anything that is a reminder of threat or trauma. Just because you have been in a car accident, for instance, doesn’t mean you’ll be hyper-vigilant only for screeching brakes or beeping horns.

A month after my psychologist told me I had PTSD, I called her, wondering if I should go to the emergency room because my toe was red – I thought I had a life-threatening infection (I’m a nurse, and sometimes nurses know too much). I wouldn’t sleep in my bedroom on the third floor of my apartment because I was afraid of dying in a fire (I worked as a burn nurse years ago).

Months later, when shopping at an outside market with my father, I suddenly felt short of breath and couldn’t swallow. I told him he needed to drive me to the emergency room because I thought I was having a heart attack. I was a physically fit, non-smoking, lover-of-veggies thirty seven year old. I was not at risk for a heart attack. I called 911 three more times in the next few months, because I thought I was having allergic reaction: first to chocolate, then shellfish, then a bug bite. Miraculously, each time the EMT’s arrived, my rapid pulse slowed and my quivering body relaxed. I was suffering from panic attacks.

Before the accident, I had been known for my calm demeanor, and my no-worry attitude in my family. When working in the neonatal intensive care unit, I had been known for my in-control, I-can-handle-this disposition, even when a baby’s heart rate plummeted to near zero. After the accident, I felt as if there was a circuit breaker inside my brain that tripped at random moments, sending sparks into my nervous system. I didn’t know exactly where the breaker was located, or how to stop it from tripping. I reasoned that my brain had been re-wired. My reasoning was accurate – recently, I learned that researchers have found differences in the structure and circuitry of the brain between those with PTSD and those without it.

http://www.ptsd.va.gov/professional/treatment/overview/clinicians-guide-to-medications-for-ptsd.asp